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Furthermore, glycogen metabolism supports DC maturation, inflammatory cytokine production, and priming of the nucleotide-binding area, leucine-rich-containing household, pyrin domain-containing-three (NLRP3) inflammasome in response to each TLR- and CLR-mediated activation. These data assist a mannequin in which different lessons of innate immune receptors functionally converge in their requirement for glycogen-dependent glycolysis to metabolically assist early DC activation. These research provide new perception into how DC immune effector function is metabolically regulated in response to various inflammatory stimuli.<br> <br>Ketone levels frequently rise. 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Latest revision as of 01:49, 22 October 2025


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